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dc.contributor.CRUESPUniversidade Estadual de Campinaspt_BR
dc.typeArtigo de periódicopt_BR
dc.titleNon-effect of p22-phox-930A/G polymorphism on end-organ damage in Brazilian hypertensive patientspt_BR
dc.contributor.authorSales, MLpt_BR
dc.contributor.authorFerreira, MCSpt_BR
dc.contributor.authorLeme, CApt_BR
dc.contributor.authorVelloso, LApt_BR
dc.contributor.authorGallani, MCJpt_BR
dc.contributor.authorColombo, RCRpt_BR
dc.contributor.authorFranchini, KGpt_BR
dc.contributor.authorNadruz, Wpt_BR
unicamp.author.emailwilnj@fcm.unicamp.brpt_BR
unicamp.authorUniv Estadual Campinas, Dept Internal Med, Sch Med, Campinas, SP, Brazil Univ Estadual Campinas, Sch Nursing, Campinas, SP, Brazilpt_BR
dc.subject.wosNadph Oxidase Activitypt_BR
dc.subject.wosExpressionpt_BR
dc.subject.wosProteinpt_BR
dc.description.abstractThe p22-phox subunit is an essential component of NAD(P)H oxidase enzymatic complex, which is considered the major source of oxidative stress products in the cardiovascular system. 1,2 The -930G allele of p22-phox has been associated with higher promoter activity, increased NAD( P) H oxidase-mediated oxidative stress and hypertension. 3,4 We recently reported that left ventricular hypertrophy is accompanied by increased myocardial p22-phox expression in aortic-banded rats, suggesting that this protein might be involved in hypertensive cardiac hypertrophy.(2) Thus, the aim of the present report was to investigate the role of p22-phox -930A/G polymorphism on end-organ damage in hypertensive patients.pt
dc.relation.ispartofJournal Of Human Hypertensionpt_BR
dc.relation.ispartofabbreviationJ. Hum. Hypertens.pt_BR
dc.publisher.cityLondonpt_BR
dc.publisher.countryInglaterrapt_BR
dc.publisherNature Publishing Grouppt_BR
dc.date.issued2007pt_BR
dc.date.monthofcirculationJUNpt_BR
dc.identifier.citationJournal Of Human Hypertension. Nature Publishing Group, v. 21, n. 6, n. 504, n. 506, 2007.pt_BR
dc.language.isoenpt_BR
dc.description.volume21pt_BR
dc.description.issuenumber6pt_BR
dc.description.firstpage504pt_BR
dc.description.lastpage506pt_BR
dc.rightsfechadopt_BR
dc.sourceWeb of Sciencept_BR
dc.identifier.issn0950-9240pt_BR
dc.identifier.wosidWOS:000246799500014pt_BR
dc.identifier.doi10.1038/sj.jhh.1002170pt_BR
dc.date.available2014-11-14T14:34:56Z
dc.date.available2015-11-26T16:06:58Z-
dc.date.accessioned2014-11-14T14:34:56Z
dc.date.accessioned2015-11-26T16:06:58Z-
dc.description.provenanceMade available in DSpace on 2014-11-14T14:34:56Z (GMT). No. of bitstreams: 1 WOS000246799500014.pdf: 69405 bytes, checksum: ca40aaf0f68f9fd97d5fb88d01340112 (MD5) Previous issue date: 2007en
dc.description.provenanceMade available in DSpace on 2015-11-26T16:06:58Z (GMT). No. of bitstreams: 2 WOS000246799500014.pdf: 69405 bytes, checksum: ca40aaf0f68f9fd97d5fb88d01340112 (MD5) WOS000246799500014.pdf.txt: 11737 bytes, checksum: fa94e1e1bb3e276adfdd26606d19cf6b (MD5) Previous issue date: 2007en
dc.identifier.urihttp://www.repositorio.unicamp.br/jspui/handle/REPOSIP/61977pt_BR
dc.identifier.urihttp://www.repositorio.unicamp.br/handle/REPOSIP/61977
dc.identifier.urihttp://repositorio.unicamp.br/jspui/handle/REPOSIP/61977-
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